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Commercial Name: Lyrica Principal Ingredient: PregabalinUtilization: Relieves: Pain ManagementAvailable Dosage: 30 mg, 60 mg and 90 mg

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In animal models of nerve damage, pregabalin has been shown to reduce calciumdependent release of pro-nociceptive neurotransmitters in the spinal cord, possibly by disrupting alpha2-delta lyrica containing-calcium channel trafficking and/or reducing calcium currents .
Evidence from other animal models of nerve damage and persistent pain suggest the anti-nociceptive activities of pregabalin may also be mediated through interactions with descending noradrenergic and serotonergic pathways originating from the brainstem that modulate pain transmission in the While pregabalin is a structural derivative of the inhibitory neurotransmitter gamma aminobutyric acid (GABA), it does not lyrica bind directly to GABAA, GABAB, or benzodiazepine receptors, does not augment GABAA responses in cultured neurons, does not alter rat brain GABA concentration or have acute effects on GABA uptake or degradation .
However, in cultured neurons prolonged application of pregabalin increases the density of GABA transporter protein and increases the rate lyrica extrapyramidal side effects lyrica anderson mom singing lyrica side effects numbness tingling of functional GABA transport .
Pregabalin does not block sodium channels, is not active at opiate receptors, and does not alter cyclooxygenase enzyme lyrica activity .
It is inactive at serotonin and dopamine receptors and does not inhibit lyrical choreography lyrica coupon for medicare patients lyrical prose definition lyrical quotes dopamine, serotonin, or noradrenaline reuptake .
3 Pharmacokinetics Pregabalin is well absorbed after oral administration, is eliminated lyrica largely by renal excretion, and has an elimination half-life of about 6 hours .

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